Therapy resistance: opportunities created by adaptive responses to targeted therapies in cancer | Nature Reviews Cancer
The plasticity of DNA replication forks in response to clinically relevant genotoxic stress | Nature Reviews Molecular Cell Biology
Frontiers | UFL1, a UFMylation E3 ligase, plays a crucial role in multiple cellular stress responses
Frontiers | DNA Damage Tolerance Pathways in Human Cells: A Potential Therapeutic Target
Frontiers | A Survey of Essential Genome Stability Genes Reveals That Replication Stress Mitigation Is Critical for Peri-Implantation Embryogenesis
Frontiers | Single-Molecule Techniques to Study Chromatin
DNA double-strand break repair-pathway choice in somatic mammalian cells | Nature Reviews Molecular Cell Biology
Two replication fork remodeling pathways generate nuclease substrates for distinct fork protection factors | Science Advances
To skip or not to skip: choosing repriming to tolerate DNA damage - ScienceDirect
Cell cycle control in cancer | Nature Reviews Molecular Cell Biology
Ubiquitylation at Stressed Replication Forks: Mechanisms and Functions: Trends in Cell Biology
Pathogens | Free Full-Text | Tissue Pathogens and Cancers: A Review of Commonly Seen Manifestations in Histo- and Cytopathology
Full article: Building up and breaking down: mechanisms controlling recombination during replication
Frontiers | Replication Fork Reversal and Protection
Genes | Free Full-Text | Replication-Associated Recombinational Repair: Lessons from Budding Yeast
Leveraging the replication stress response to optimize cancer therapy | Nature Reviews Cancer
RIF1 promotes replication fork protection and efficient restart to maintain genome stability | Nature Communications
Frontiers | Rad5 participates in lesion bypass through its Rev1-binding and ubiquitin ligase domains, but not through its helicase function
Super-resolution visualization of distinct stalled and broken replication fork structures | PLOS Genetics
Heterogeneity in tuberculosis | Nature Reviews Immunology
WRN helicase safeguards deprotected replication forks in BRCA2-mutated cancer cells | Nature Communications
Kai Neelsen – Senior Medical Writer – nspm ltd | LinkedIn
Smc5/6 functions with Sgs1-Top3-Rmi1 to complete chromosome replication at natural pause sites | Nature Communications
Replication-Coupled DNA Repair - ScienceDirect
